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Therefore, it absolutely was affordable that the system of inhibition of major WT1 isoform-mediated transcriptional activation of Bcl-xL and Bcl-2 by Ex4a(+)WT1 isoform may be level of competition with important WT1 isoform for interacting along with the transcriptional co-activators aside from WT1 alone, resulting while in the abrogation of important WT1 isoform-mediated transcriptional action in

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